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Vels of viral particles, we speculate that average levels of host-viral chimeric transcripts in these cells exceed those in the two J-Lat cell lines. These results thus suggest that TI plays a key role in primary CD4+ T cells. In J-Lat 9.2 and 15.4 cells, the host-viral chimeric transcripts terminated at the pA site in the 5'LTR, but transcripts initiating at the host promoter and terminating at t
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Lpropane-1,3-diamine. Acknowledgements We especially thank the patients and surgeons, pathologists, and internists for their assistance in collecting tumor tissues and patients' clinical follow-up data. We thank Joan Bolt, Marion Meijer, Mieke Timmermans, Anita Trapman, and Wendy van der Smissen for their excellent technical support. This work was financially supported by VICI grant 918-66-615 (aw
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O OH O O O OH OO OO_scabellones AStructure AntimalarialO OH O O O OH OO OO_scabellones AStructure Anti
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Everse transcriptase inhibitors (NNRTIs). RMP decreases the plasma concentration of protease inhibitors (PIs) to subtherapeutic levels when the PIs are given at standard doses [15?8]. Coadministration of the PI at higher doses or super-boosting the PI with higher doses of ritonavir (RTV, or r) may result in unacceptably high rates of liver toxicity [18?1]. To complicate things further, risk of tox
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Ce Council: Dutch guidelines for pharmacoeconomic research. Amstelveen: Ziekenfondsraad; 1999.doi:10.1186/1471-2261-14-1 Cite this article as: van Riet et al.: Strategy to recognize and initiate treatment of chronic heart failure in primary care (STRETCH): a cluster randomized trial. BMC Cardiovascular Disorders 2014 14:1.Submit your next manuscript to BioMed Central and take full advantage of:?Co
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Exposed to autoradiography film. High-mannose moieties were preserved only in untreated virus confirming that PNGase F and endo H effectively cleaved high-mannose residues on GPs of HIV-EBOV virion-like particles. (EPS) Figure S4 Thermolysin treatment of HIV-EBOV GP abrogates enhancement of infection by rhMBL in a thermolysin-concentration dependent manner. We preincubated HIV-EBOV GP virion-like
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