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Channels at glutamatergic synapses (Maximov Bezprozvanny 2002). In the postsynaptic compartment, NL1 may bind through cytoplasmic PDZ-domain interactions to PSD95 and thereby recruit NMDA-Rs to glutamatergic synapses (Irie et al. 1997). Although the relevance of PSD95 or other scaffolding molecules for the NL1-mediated synaptic recruitment of NMDA-Rs remains to be tested, a conceptually similar

Differences that feed on existing inequalities in the society, such as those found in gender [5,6]. While sex can be defined as the biological distinction between men and women [7], Schur (p. 10) describes gender as "the socio cultural and psychological shaping, patterning and evaluation of male and female behaviour" [8]. Butler (p. 42) similarly noted that gender is a mechanism by which notions o

Tion-inducing activities of ESR1 [1,2]. Unraveling the precise role of DC-SCRIPT in the complex genomic and non-genomic interplay between ESR1, ESR2, and their isoforms [21-23] might turn out to be rewarding for elucidating the `yin-yang' role of these factors in breast cancer. As DC-SCRIPT can inhibit ERa and PR activity, a second aim of the study was to address whether DCSCRIPT affects the respo